Frontiers Agomelatine Prevents Amyloid Plaque Deposition, Tau Phosphorylation, and Neuroinflammation in APP/PS1 Mice

Immune-mediated platelet depletion augments Alzheimer's disease

Frontiers Agomelatine Prevents Amyloid Plaque Deposition, Tau

Plasmon-Activated Water Reduces Amyloid Burden and Improves Memory

Frontiers Overnutrition Induced Cognitive Impairment: Insulin Resistance, Gut-Brain Axis, and Neuroinflammation

Lack of Tau Proteins Rescues Neuronal Cell Death and Decreases

Frontiers Neuropeptide S Ameliorates Cognitive Impairment of APP/PS1 Transgenic Mice by Promoting Synaptic Plasticity and Reducing Aβ Deposition

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Loss of presenilin function enhances tau phosphorylation and

Frontiers Plaque-Associated Oligomeric Amyloid-Beta Drives Early

PDF) Regulation of Melatonin and Neurotransmission in Alzheimer's

dhcr24 - List of Frontiers' open access articles

Locomotor Hyperactivity in the Early-Stage Alzheimer's Disease

Neurophysiological effects of human-derived pathological tau

Agomelatine improves memory and learning impairments in a rat